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It promotes healing by supporting cell migration to injured areas and aiding the regeneration of damaged tissue, particularly in muscles, tendons, and ligaments
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Alterations resulting from METex14 mutations are marked by the partial removal of the parallel membrane configuration or hindered attachment to the E3 ubiquitin-protein ligase CBL, which impedes MET protein ubiquitination and self-degradation, thereby extending the activation of subsequent signals [23, 24]
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